hnRNP A1/A2 and Sam68 collaborate with SRSF10 to control the alternative splicing response to oxaliplatin-mediated DNA damage

Little is known about how RNA binding proteins cooperate to control splicing, and how stress pathways reconfigure these assemblies to alter splice site selection. We have shown previously that SRSF10 plays an important role in the Bcl-x splicing response to DNA damage elicited by oxaliplatin in 293...

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Detalles Bibliográficos
Autores principales: Cloutier, Alexandre, Shkreta, Lulzim, Toutant, Johanne, Durand, Mathieu, Thibault, Philippe, Chabot, Benoit
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2018
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5797138/
https://www.ncbi.nlm.nih.gov/pubmed/29396485
http://dx.doi.org/10.1038/s41598-018-20360-x

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5797138/
https://www.ncbi.nlm.nih.gov/pubmed/29396485
http://dx.doi.org/10.1038/s41598-018-20360-x

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