Cx26 drives self-renewal in triple-negative breast cancer via interaction with NANOG and focal adhesion kinase

Tumors adapt their phenotypes during growth and in response to therapies through dynamic changes in cellular processes. Connexin proteins enable such dynamic changes during development, and their dysregulation leads to disease states. The gap junction communication channels formed by connexins have...

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Detalles Bibliográficos
Autores principales: Thiagarajan, Praveena S., Sinyuk, Maksim, Turaga, Soumya M., Mulkearns-Hubert, Erin E., Hale, James S., Rao, Vinay, Demelash, Abeba, Saygin, Caner, China, Arnab, Alban, Tyler J., Hitomi, Masahiro, Torre-Healy, Luke A., Alvarado, Alvaro G., Jarrar, Awad, Wiechert, Andrew, Adorno-Cruz, Valery, Fox, Paul L., Calhoun, Benjamin C., Guan, Jun-Lin, Liu, Huiping, Reizes, Ofer, Lathia, Justin D.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2018
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5805730/
https://www.ncbi.nlm.nih.gov/pubmed/29422613
http://dx.doi.org/10.1038/s41467-018-02938-1

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5805730/
https://www.ncbi.nlm.nih.gov/pubmed/29422613
http://dx.doi.org/10.1038/s41467-018-02938-1

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