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Soluble Aβ(1–42) increases the heterogeneity in synaptic vesicle pool size among synapses by suppressing intersynaptic vesicle sharing
Growing evidence has indicated that prefibrillar form of soluble amyloid beta (sAβ(1–42)) is the major causative factor in the synaptic dysfunction associated with AD. The molecular changes leading to presynaptic dysfunction caused by sAβ(1–42), however, still remains elusive. Recently, we found tha...
| Autores principales: | , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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BioMed Central
2018
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5819658/ https://www.ncbi.nlm.nih.gov/pubmed/29463281 http://dx.doi.org/10.1186/s13041-018-0353-z |