Cargando…
Human immunodeficiency virus Tat impairs mitochondrial fission in neurons
Human immunodeficiency virus-1 (HIV) infection of the central nervous system promotes neuronal injury that culminates in HIV-associated neurocognitive disorders. Viral proteins, including transactivator of transcription (Tat), have emerged as leading candidates to explain HIV-mediated neurotoxicity,...
Autores principales: | Rozzi, Summer J., Avdoshina, Valeria, Fields, Jerel A., Mocchetti, Italo |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5841280/ https://www.ncbi.nlm.nih.gov/pubmed/29531805 http://dx.doi.org/10.1038/s41420-017-0013-6 |
Ejemplares similares
-
Recent Advances in the Molecular and Cellular Mechanisms of gp120-Mediated Neurotoxicity
por: Avdoshina, Valeria, et al.
Publicado: (2022) -
Helix-A peptide prevents gp120-mediated neuronal loss
por: Avdoshina, Valeria, et al.
Publicado: (2019) -
Reversal of Cognitive Impairment in gp120 Transgenic Mice by the Removal of the p75 Neurotrophin Receptor
por: Speidell, Andrew, et al.
Publicado: (2019) -
Histone deacetylase 6 inhibition rescues axonal transport impairments and prevents the neurotoxicity of HIV-1 envelope protein gp120
por: Wenzel, Erin D., et al.
Publicado: (2019) -
Prevention of HIV-1 TAT Protein-Induced Peripheral Neuropathy and Mitochondrial Disruption by the Antimuscarinic Pirenzepine
por: Han, May Madi, et al.
Publicado: (2021)