Chronic p38 mitogen-activated protein kinase inhibition improves vascular function and remodeling in angiotensin II-dependent hypertension

INTRODUCTION: An excess of angiotensin II (Ang II) causes hypertension and vascular injury. Activation of mitogen-activated protein kinase p38 (p38-MAPK) plays a substantial role in Ang II-dependent organ damage. Recently, we showed that p38-MAPK activation regulates the pressor response to Ang II....

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Detalles Bibliográficos
Autores principales: Potthoff, SA, Stamer, S, Grave, K, Königshausen, E, Sivritas, SH, Thieme, M, Mori, Y, Woznowski, M, Rump, LC, Stegbauer, J
Formato: Online Artículo Texto
Lenguaje:English
Publicado: SAGE Publications 2016
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5843849/
https://www.ncbi.nlm.nih.gov/pubmed/27407119
http://dx.doi.org/10.1177/1470320316653284

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5843849/
https://www.ncbi.nlm.nih.gov/pubmed/27407119
http://dx.doi.org/10.1177/1470320316653284

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