circHECTD1 promotes the silica-induced pulmonary endothelial–mesenchymal transition via HECTD1

Excessive proliferation and migration of fibroblasts contribute to pulmonary fibrosis in silicosis, and both epithelial cells and endothelial cells participate in the accumulation of fibroblasts via the epithelial–mesenchymal transition (EMT) and the endothelial–mesenchymal transition (EndMT), respe...

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Detalles Bibliográficos
Autores principales: Fang, Shencun, Guo, Huifang, Cheng, Yusi, Zhou, Zewei, Zhang, Wei, Han, Bing, Luo, Wei, Wang, Jing, Xie, Weiping, Chao, Jie
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2018
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5852113/
https://www.ncbi.nlm.nih.gov/pubmed/29540674
http://dx.doi.org/10.1038/s41419-018-0432-1

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5852113/
https://www.ncbi.nlm.nih.gov/pubmed/29540674
http://dx.doi.org/10.1038/s41419-018-0432-1

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