RIPK3-deficient mice were not protected from nephrotoxic nephritis

BACKGROUND: Necrotizing glomerular lesions are a feature of severe glomerulonephritis. Unlike apoptosis, cellular necrosis has the potential to release damage-associated proteins into the microenvironment, thereby potentiating inflammation. Until recently necrosis was thought to be an unregulated ce...

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Detalles Bibliográficos
Autores principales: Hill, N. R., Cook, H. T., Pusey, C. D., Tarzi, R. M.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2018
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5853157/
https://www.ncbi.nlm.nih.gov/pubmed/29540162
http://dx.doi.org/10.1186/s12882-018-0850-4

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5853157/
https://www.ncbi.nlm.nih.gov/pubmed/29540162
http://dx.doi.org/10.1186/s12882-018-0850-4

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