GIT1 regulates synaptic structural plasticity underlying learning

The signaling scaffold protein GIT1 is expressed widely throughout the brain, but its function in vivo remains elusive. Mice lacking GIT1 have been proposed as a model for attention deficit-hyperactivity disorder, due to alterations in basal locomotor activity as well as paradoxical locomotor suppre...

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Detalles Bibliográficos
Autores principales: Martyn, Amanda C., Toth, Krisztian, Schmalzigaug, Robert, Hedrick, Nathan G., Rodriguiz, Ramona M., Yasuda, Ryohei, Wetsel, William C., Premont, Richard T.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2018
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5858814/
https://www.ncbi.nlm.nih.gov/pubmed/29554125
http://dx.doi.org/10.1371/journal.pone.0194350

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5858814/
https://www.ncbi.nlm.nih.gov/pubmed/29554125
http://dx.doi.org/10.1371/journal.pone.0194350

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