Cargando…
GIT1 regulates synaptic structural plasticity underlying learning
The signaling scaffold protein GIT1 is expressed widely throughout the brain, but its function in vivo remains elusive. Mice lacking GIT1 have been proposed as a model for attention deficit-hyperactivity disorder, due to alterations in basal locomotor activity as well as paradoxical locomotor suppre...
Autores principales: | Martyn, Amanda C., Toth, Krisztian, Schmalzigaug, Robert, Hedrick, Nathan G., Rodriguiz, Ramona M., Yasuda, Ryohei, Wetsel, William C., Premont, Richard T. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5858814/ https://www.ncbi.nlm.nih.gov/pubmed/29554125 http://dx.doi.org/10.1371/journal.pone.0194350 |
Ejemplares similares
-
GIT2 Acts as a Systems-Level Coordinator of Neurometabolic Activity and Pathophysiological Aging
por: Martin, Bronwen, et al.
Publicado: (2016) -
Brain-Specific Deletion of GIT1 Impairs Cognition and Alters Phosphorylation of Synaptic Protein Networks Implicated in Schizophrenia Susceptibility
por: Fass, Daniel M., et al.
Publicado: (2022) -
The G protein biased serotonin 5-HT 2A receptor agonist lisuride exerts anti-depressant drug-like activities in mice
por: Pogorelov, Vladimir M., et al.
Publicado: (2023) -
The G protein biased serotonin 5-HT2A receptor agonist lisuride exerts anti-depressant drug-like activities in mice
por: Pogorelov, Vladimir M., et al.
Publicado: (2023) -
Version control with Git and GitHub
por: Magana, Alex, et al.
Publicado: (2018)