HSV-1-induced disruption of transcription termination resembles a cellular stress response but selectively increases chromatin accessibility downstream of genes

Lytic herpes simplex virus 1 (HSV-1) infection triggers disruption of transcription termination (DoTT) of most cellular genes, resulting in extensive intergenic transcription. Similarly, cellular stress responses lead to gene-specific transcription downstream of genes (DoG). In this study, we perfor...

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Detalles Bibliográficos
Autores principales: Hennig, Thomas, Michalski, Marco, Rutkowski, Andrzej J., Djakovic, Lara, Whisnant, Adam W., Friedl, Marie-Sophie, Jha, Bhaskar Anand, Baptista, Marisa A. P., L’Hernault, Anne, Erhard, Florian, Dölken, Lars, Friedel, Caroline C.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2018
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5886697/
https://www.ncbi.nlm.nih.gov/pubmed/29579120
http://dx.doi.org/10.1371/journal.ppat.1006954

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5886697/
https://www.ncbi.nlm.nih.gov/pubmed/29579120
http://dx.doi.org/10.1371/journal.ppat.1006954

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