Ketamine ameliorates oxidative stress-induced apoptosis in experimental traumatic brain injury via the Nrf2 pathway

BACKGROUND: Ketamine can act as a multifunctional neuroprotective agent by inhibiting oxidative stress, cellular dysfunction, and apoptosis. Although it has been proven to be effective in various neurologic disorders, the mechanism of the treatment of traumatic brain injury (TBI) is not fully unders...

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Detalles Bibliográficos
Autores principales: Liang, Jinwei, Wu, Shanhu, Xie, Wenxi, He, Hefan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Dove Medical Press 2018
Materias:
Original Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5907785/
https://www.ncbi.nlm.nih.gov/pubmed/29713142
http://dx.doi.org/10.2147/DDDT.S160046

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5907785/
https://www.ncbi.nlm.nih.gov/pubmed/29713142
http://dx.doi.org/10.2147/DDDT.S160046

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