FAM35A associates with REV7 and modulates DNA damage responses of normal and BRCA1‐defective cells

To exploit vulnerabilities of tumors, it is urgent to identify associated defects in genome maintenance. One unsolved problem is the mechanism of regulation of DNA double‐strand break repair by REV7 in complex with 53BP1 and RIF1, and its influence on repair pathway choice between homologous recombi...

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Detalles Bibliográficos
Autores principales: Tomida, Junya, Takata, Kei‐ichi, Bhetawal, Sarita, Person, Maria D, Chao, Hsueh‐Ping, Tang, Dean G, Wood, Richard D
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2018
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6003645/
https://www.ncbi.nlm.nih.gov/pubmed/29789392
http://dx.doi.org/10.15252/embj.201899543

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6003645/
https://www.ncbi.nlm.nih.gov/pubmed/29789392
http://dx.doi.org/10.15252/embj.201899543

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