Trpc6 inactivation confers protection in a model of severe nephrosis in rats

ABSTRACT: Mutations in canonical transient receptor potential-6 (TRPC6) channels give rise to rare familial forms of focal and segmental glomerulosclerosis (FSGS). Here we examined a possible role for TRPC6 in the progression of chronic puromycin aminonucleoside (PAN) nephrosis in Sprague-Dawley rat...

Descripción completa

Detalles Bibliográficos
Autores principales: Kim, Eun Young, Yazdizadeh Shotorbani, Parisa, Dryer, Stuart E.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer Berlin Heidelberg 2018
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6015123/
https://www.ncbi.nlm.nih.gov/pubmed/29785489
http://dx.doi.org/10.1007/s00109-018-1648-3

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6015123/
https://www.ncbi.nlm.nih.gov/pubmed/29785489
http://dx.doi.org/10.1007/s00109-018-1648-3

Ejemplares similares