Cargando…
Mutually exclusive acetylation and ubiquitylation of the splicing factor SRSF5 control tumor growth
Most tumor cells take up more glucose than normal cells. Splicing dysregulation is one of the molecular hallmarks of cancer. However, the role of splicing factor in glucose metabolism and tumor development remains poorly defined. Here, we show that upon glucose intake, the splicing factor SRSF5 is s...
Autores principales: | Chen, Yuhan, Huang, Qingyang, Liu, Wen, Zhu, Qiong, Cui, Chun-Ping, Xu, Liang, Guo, Xing, Wang, Ping, Liu, Jingwen, Dong, Guanglong, Wei, Wenyi, Liu, Cui Hua, Feng, Zhichun, He, Fuchu, Zhang, Lingqiang |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6018636/ https://www.ncbi.nlm.nih.gov/pubmed/29942010 http://dx.doi.org/10.1038/s41467-018-04815-3 |
Ejemplares similares
-
Mutually exclusive acetylation and ubiquitylation among enzymes involved in glucose metabolism
por: Ande, Sudharsana Rao, et al.
Publicado: (2013) -
Splicing factor Srsf5 deletion disrupts alternative splicing and causes noncompaction of ventricular myocardium
por: Zhang, Xiaoli, et al.
Publicado: (2021) -
Dynamic ubiquitylation of Sox2 regulates proteostasis and governs neural progenitor cell differentiation
por: Cui, Chun-Ping, et al.
Publicado: (2018) -
Author Correction: Dynamic ubiquitylation of Sox2 regulates proteostasis and governs neural progenitor cell differentiation
por: Cui, Chun-Ping, et al.
Publicado: (2019) -
The landscape of human mutually exclusive splicing
por: Hatje, Klas, et al.
Publicado: (2017)