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Oxidation of KCNB1 channels in the human brain and in mouse model of Alzheimer’s disease
Oxidative modification of the voltage-gated K(+) channel subfamily B member 1 (KCNB1, Kv2.1) is emerging as a mechanism of neuronal vulnerability potentially capable of affecting multiple conditions associated with oxidative stress, from normal aging to neurodegenerative disease. In this study we re...
| Autores principales: | , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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Nature Publishing Group UK
2018
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6062629/ https://www.ncbi.nlm.nih.gov/pubmed/30050035 http://dx.doi.org/10.1038/s41419-018-0886-1 |