Atg9a deficiency causes axon-specific lesions including neuronal circuit dysgenesis

Conditional knockout mice for Atg9a, specifically in brain tissue, were generated to understand the roles of ATG9A in the neural tissue cells. The mice were born normally, but half of them died within one wk, and none lived beyond 4 wk of age. SQSTM1/p62 and NBR1, receptor proteins for selective aut...

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Detalles Bibliográficos
Autores principales: Yamaguchi, Junji, Suzuki, Chigure, Nanao, Tomohisa, Kakuta, Soichirou, Ozawa, Kentarou, Tanida, Isei, Saitoh, Tatsuya, Sunabori, Takehiko, Komatsu, Masaaki, Tanaka, Keiji, Aoki, Shigeki, Sakimura, Kenji, Uchiyama, Yasuo
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Taylor & Francis 2018
Materias:
Research Paper - Basic Science
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6070006/
https://www.ncbi.nlm.nih.gov/pubmed/28513333
http://dx.doi.org/10.1080/15548627.2017.1314897

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6070006/
https://www.ncbi.nlm.nih.gov/pubmed/28513333
http://dx.doi.org/10.1080/15548627.2017.1314897

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