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Atg9a deficiency causes axon-specific lesions including neuronal circuit dysgenesis
Conditional knockout mice for Atg9a, specifically in brain tissue, were generated to understand the roles of ATG9A in the neural tissue cells. The mice were born normally, but half of them died within one wk, and none lived beyond 4 wk of age. SQSTM1/p62 and NBR1, receptor proteins for selective aut...
Autores principales: | Yamaguchi, Junji, Suzuki, Chigure, Nanao, Tomohisa, Kakuta, Soichirou, Ozawa, Kentarou, Tanida, Isei, Saitoh, Tatsuya, Sunabori, Takehiko, Komatsu, Masaaki, Tanaka, Keiji, Aoki, Shigeki, Sakimura, Kenji, Uchiyama, Yasuo |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Taylor & Francis
2018
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6070006/ https://www.ncbi.nlm.nih.gov/pubmed/28513333 http://dx.doi.org/10.1080/15548627.2017.1314897 |
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