PM(2.5) downregulates miR-194-3p and accelerates apoptosis in cigarette-inflamed bronchial epithelium by targeting death-associated protein kinase 1

BACKGROUND: Persistent exposure to cigarette smoke or biomass fuels induces oxidative stress and apoptosis in bronchial epithelium, which is one of the most important pathogenic mechanisms of chronic obstructive pulmonary disease (COPD). Fine particulate matter (PM(2.5)) is an aggravating risk facto...

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Detalles Bibliográficos
Autores principales: Zhou, Tianyu, Zhong, Yijue, Hu, Yan, Sun, Chao, Wang, Yunxia, Wang, Guangfa
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Dove Medical Press 2018
Materias:
Original Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6078088/
https://www.ncbi.nlm.nih.gov/pubmed/30122914
http://dx.doi.org/10.2147/COPD.S168629

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6078088/
https://www.ncbi.nlm.nih.gov/pubmed/30122914
http://dx.doi.org/10.2147/COPD.S168629

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