Genetic Deletion of PGF(2α)-FP Receptor Exacerbates Brain Injury Following Experimental Intracerebral Hemorrhage

Background: The release of inflammatory molecules such as prostaglandins (e.g., PGF(2α)) is associated with brain damage following an intracerebral hemorrhagic (ICH) stroke; however, the role of PGF(2α) and its cognate FP receptor in ICH remains unclear. This study focused on investigating the role...

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Detalles Bibliográficos
Autores principales: Mohan, Shekher, Koller, Emily J., Fazal, Jawad A., De Oliveria, Gabriela, Pawlowicz, Anna I., Doré, Sylvain
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2018
Materias:
Neuroscience
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6134069/
https://www.ncbi.nlm.nih.gov/pubmed/30233287
http://dx.doi.org/10.3389/fnins.2018.00556

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6134069/
https://www.ncbi.nlm.nih.gov/pubmed/30233287
http://dx.doi.org/10.3389/fnins.2018.00556

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