Expression of calcineurin, calpastatin and heat shock proteins during ischemia and reperfusion

OBJECTIVE: Calcineurin (CaN) interacts with calpains (Calpn) and causes cellular damage eventually leading to cell death. Calpastatin (Calp) is a specific Calpn inhibitor, along with CaN stimulation has been implicated in reduced cell death and self-repair. Molecular chaperones, heat shock proteins...

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Detalles Bibliográficos
Autores principales: Parameswaran, Sreejit, Sharma, Rajendra K.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2015
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6189699/
https://www.ncbi.nlm.nih.gov/pubmed/30338302
http://dx.doi.org/10.1016/j.bbrep.2015.09.016

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6189699/
https://www.ncbi.nlm.nih.gov/pubmed/30338302
http://dx.doi.org/10.1016/j.bbrep.2015.09.016

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