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HIV-1 Vpr Reprograms CLR4(DCAF1) E3 Ubiquitin Ligase to Antagonize Exonuclease 1-Mediated Restriction of HIV-1 Infection
Viral accessory proteins hijack host cell E3 ubiquitin ligases to antagonize innate/intrinsic defenses and thereby provide a more permissive environment for virus replication. Human immunodeficiency virus type 1 (HIV-1) accessory protein Vpr reprograms CRL4(DCAF1) E3 to antagonize select postreplica...
Autores principales: | Yan, Junpeng, Shun, Ming-Chieh, Hao, Caili, Zhang, Yi, Qian, Juan, Hrecka, Kasia, DeLucia, Maria, Monnie, Christina, Ahn, Jinwoo, Skowronski, Jacek |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
American Society for Microbiology
2018
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6199497/ https://www.ncbi.nlm.nih.gov/pubmed/30352932 http://dx.doi.org/10.1128/mBio.01732-18 |
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