eNOS expression and NO release during hypoxia is inhibited by miR-200b in human endothelial cells

The nitric oxide (NO) secreted by vascular endothelium is required for the maintenance of cardiovascular homeostasis. Diminished release of NO generated by endothelial NO synthase contributes to endothelial dysfunction. Hypoxia and ischemia reduce endothelial eNOS expression via posttranscriptional...

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Detalles Bibliográficos
Autores principales: Janaszak-Jasiecka, Anna, Siekierzycka, Anna, Bartoszewska, Sylwia, Serocki, Marcin, Dobrucki, Lawrence W., Collawn, James F., Kalinowski, Leszek, Bartoszewski, Rafal
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer Netherlands 2018
Materias:
Brief Communication
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6208887/
https://www.ncbi.nlm.nih.gov/pubmed/29737439
http://dx.doi.org/10.1007/s10456-018-9620-y

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6208887/
https://www.ncbi.nlm.nih.gov/pubmed/29737439
http://dx.doi.org/10.1007/s10456-018-9620-y

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