Cargando…
How does glucagon‐like peptide 1 stimulate human β‐cell proliferation? A lesson from islet graft experiments
The incidence of type 2 diabetes increases with age. The age‐dependent decline in functional β‐cell mass contributes to the increased risk of onset of diabetes, reflecting the central role of pancreatic β‐cells in glucose homeostasis. Indeed, the replication rate of human and rodent β‐cells is known...
Autor principal: | Fujitani, Yoshio |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6215945/ https://www.ncbi.nlm.nih.gov/pubmed/29770600 http://dx.doi.org/10.1111/jdi.12861 |
Ejemplares similares
-
Incretin concept revised: The origin of the insulinotropic function of glucagon‐like peptide‐1 – the gut, the islets or both?
por: Yabe, Daisuke, et al.
Publicado: (2017) -
Is ghrelin a glucagon‐like peptide‐1 secretagogue?
por: Ueno, Hiroaki, et al.
Publicado: (2015) -
Clinical application of glucagon-like peptide-1 receptor agonists in cardiovascular disease: lessons from recent clinical cardiovascular outcomes trials
por: Tanaka, Atsushi, et al.
Publicado: (2018) -
Glucagon‐like peptide‐1 receptor agonists and their effects on weight reduction
por: Shin, Shyi‐Jang
Publicado: (2012) -
Regulation of glucagon‐like peptide‐1 sensitivity by gut microbiota dysbiosis
por: Yamane, Shunsuke, et al.
Publicado: (2017)