Müller glial dysfunction during diabetic retinopathy in rats is reduced by the acrolein-scavenging drug, 2-hydrazino-4,6-dimethylpyrimidine

AIMS/HYPOTHESIS: Recent studies suggest that abnormal function in Müller glial cells plays an important role in the pathogenesis of diabetic retinopathy. This is associated with the selective accumulation of the acrolein-derived advanced lipoxidation end-product, N(ε)-(3-formyl-3,4-dehydropiperidino...

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Detalles Bibliográficos
Autores principales: McDowell, Rosemary E., Barabas, Peter, Augustine, Josy, Chevallier, Olivier, McCarron, Philip, Chen, Mei, McGeown, J. Graham, Curtis, Tim M.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer Berlin Heidelberg 2018
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6223850/
https://www.ncbi.nlm.nih.gov/pubmed/30112688
http://dx.doi.org/10.1007/s00125-018-4707-y

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6223850/
https://www.ncbi.nlm.nih.gov/pubmed/30112688
http://dx.doi.org/10.1007/s00125-018-4707-y

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