Pretreatment with Relaxin Does Not Restore NO-Mediated Modulation of Calcium Signal in Coronary Endothelial Cells Isolated from Spontaneously Hypertensive Rats

We demonstrated that in coronary endothelial cells (RCEs) from normotensive Wistar Kyoto rats (WKY), the hormone relaxin (RLX) increases NO production and reduces calcium transients by a NO-related mechanism. Since an impairment of the NO pathway has been described in spontaneously hypertensive rats...

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Detalles Bibliográficos
Autores principales: Nistri, Silvia, Di Cesare Mannelli, Lorenzo, Ghelardini, Carla, Zanardelli, Matteo, Bani, Daniele, Failli, Paola
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2015
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6272299/
https://www.ncbi.nlm.nih.gov/pubmed/26016544
http://dx.doi.org/10.3390/molecules20069524

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6272299/
https://www.ncbi.nlm.nih.gov/pubmed/26016544
http://dx.doi.org/10.3390/molecules20069524

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