Transcriptional upregulation of c‐MYC by AXL confers epirubicin resistance in esophageal adenocarcinoma

AXL receptor tyrosine kinase is overexpressed in esophageal adenocarcinoma (EAC) and several other types of malignancies; hence, it may be a valuable therapeutic target. Herein, we investigated the role of AXL in regulating c‐MYC expression and resistance to the chemotherapeutic agent epirubicin in...

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Detalles Bibliográficos
Autores principales: Hong, Jun, Maacha, Selma, Belkhiri, Abbes
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2018
Materias:
Research Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6275285/
https://www.ncbi.nlm.nih.gov/pubmed/30353671
http://dx.doi.org/10.1002/1878-0261.12395

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6275285/
https://www.ncbi.nlm.nih.gov/pubmed/30353671
http://dx.doi.org/10.1002/1878-0261.12395

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