Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway

BACKGROUND: Neuroinflammation is closely associated with functional outcome in subarachnoid hemorrhage (SAH) patients. Our recent study demonstrated that fluoxetine inhibited NLRP3 inflammasome activation and attenuated necrotic cell death in early brain injury after SAH, while the effects and poten...

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Detalles Bibliográficos
Autores principales: Liu, Fu-yi, Cai, Jing, Wang, Chun, Ruan, Wu, Guan, Guo-ping, Pan, Hai-zhou, Li, Jian-ru, Qian, Cong, Chen, Jing-sen, Wang, Lin, Chen, Gao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2018
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6302437/
https://www.ncbi.nlm.nih.gov/pubmed/30572907
http://dx.doi.org/10.1186/s12974-018-1388-x

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6302437/
https://www.ncbi.nlm.nih.gov/pubmed/30572907
http://dx.doi.org/10.1186/s12974-018-1388-x

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