Cargando…
The Human Cytomegalovirus U(L)38 protein drives mTOR-independent metabolic flux reprogramming by inhibiting TSC2
Human Cytomegalovirus (HCMV) infection induces several metabolic activities that are essential for viral replication. Despite the important role that this metabolic modulation plays during infection, the viral mechanisms involved are largely unclear. We find that the HCMV U(L)38 protein is responsib...
| Autores principales: | , , , |
|---|---|
| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Public Library of Science
2019
|
| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6363234/ https://www.ncbi.nlm.nih.gov/pubmed/30677091 http://dx.doi.org/10.1371/journal.ppat.1007569 |