Cargando…
Pharmacological inhibition of autophagy by 3-MA attenuates hyperuricemic nephropathy
Autophagy has been identified as a cellular process of bulk degradation of cytoplasmic components and its persistent activation is critically involved in the renal damage induced by ureteral obstruction. However, the role and underlying mechanisms of autophagy in hyperuricemic nephropathy (HN) remai...
Autores principales: | Bao, Jinfang, Shi, Yingfeng, Tao, Min, Liu, Na, Zhuang, Shougang, Yuan, Weijie |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Portland Press Ltd.
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6376616/ https://www.ncbi.nlm.nih.gov/pubmed/30293967 http://dx.doi.org/10.1042/CS20180563 |
Ejemplares similares
-
Delayed treatment with an autophagy inhibitor 3-MA alleviates the progression of hyperuricemic nephropathy
por: Shi, Yingfeng, et al.
Publicado: (2020) -
Pharmacologic targeting ERK1/2 attenuates the development and progression of hyperuricemic nephropathy in rats
por: Liu, Na, et al.
Publicado: (2017) -
Pharmacologic Targeting of BET Proteins Attenuates Hyperuricemic Nephropathy in Rats
por: Xiong, Chongxiang, et al.
Publicado: (2021) -
Blockade of Autophagy Prevents the Progression of Hyperuricemic Nephropathy Through Inhibiting NLRP3 Inflammasome-Mediated Pyroptosis
por: Hu, Yan, et al.
Publicado: (2022) -
Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Progression of Diabetic Nephropathy
por: Liu, Na, et al.
Publicado: (2017)