Differential regulation of GSK-3β in spinal dorsal horn and in hippocampus mediated by interleukin-1beta contributes to pain hypersensitivity and memory deficits following peripheral nerve injury

Accumulating evidence shows that inhibition of glycogen synthase kinase-3beta (GSK-3β) ameliorates cognitive impairments caused by a diverse array of diseases. Our previous work showed that spared nerve injury (SNI) that induces neuropathic pain causes short-term memory deficits. Here, we reported t...

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Detalles Bibliográficos
Autores principales: Mai, Chun-Lin, Wei, Xiao, Gui, Wen-Shan, Xu, Ya-Nan, Zhang, Jun, Lin, Zhen-Jia, Tan, Zhi, Meng, Ying-Tong, Li, Yong-Yong, Zhou, Li-Jun, Liu, Xian-Guo
Formato: Online Artículo Texto
Lenguaje:English
Publicado: SAGE Publications 2019
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6378430/
https://www.ncbi.nlm.nih.gov/pubmed/30632435
http://dx.doi.org/10.1177/1744806919826789

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6378430/
https://www.ncbi.nlm.nih.gov/pubmed/30632435
http://dx.doi.org/10.1177/1744806919826789

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