Apurinic/apyrimidinic endonuclease/redox factor 1 (APE1) alleviates myocardial hypoxia-reoxygenation injury by inhibiting oxidative stress and ameliorating mitochondrial dysfunction

Oxidative stress and mitochondrial dysfunction are considered to be activators of apoptosis and serve a pivotal role in the pathogenesis of myocardial ischemia-reperfusion (MI/R) injury. Apurinic/apyrimidinic endonuclease/redox factor 1 (APE1) is a multifunctional protein that processes the cellular...

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Detalles Bibliográficos
Autores principales: Hao, Jie, Du, Hong, Liu, Fan, Lu, Jing-Chao, Yang, Xiu-Chun, Cui, Wei
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2019
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6395998/
https://www.ncbi.nlm.nih.gov/pubmed/30867702
http://dx.doi.org/10.3892/etm.2019.7212

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6395998/
https://www.ncbi.nlm.nih.gov/pubmed/30867702
http://dx.doi.org/10.3892/etm.2019.7212

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