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Activation of the PERK-ATF4 pathway promotes chemo-resistance in colon cancer cells
Colon cancer is a major health problem worldwide. While chemotherapy remains a main approach for treating late-stage colon cancer patients, most, if not all, of them will develop drug resistance and die of uncontrollable disease progression eventually. Therefore, identification of mechanism of drug...
Autores principales: | , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2019
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6397152/ https://www.ncbi.nlm.nih.gov/pubmed/30824833 http://dx.doi.org/10.1038/s41598-019-39547-x |
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author | Shi, Zhong Yu, Xiaofu Yuan, Meiqin Lv, Wangxia Feng, Tingting Bai, Rui Zhong, Haijun |
author_facet | Shi, Zhong Yu, Xiaofu Yuan, Meiqin Lv, Wangxia Feng, Tingting Bai, Rui Zhong, Haijun |
author_sort | Shi, Zhong |
collection | PubMed |
description | Colon cancer is a major health problem worldwide. While chemotherapy remains a main approach for treating late-stage colon cancer patients, most, if not all, of them will develop drug resistance and die of uncontrollable disease progression eventually. Therefore, identification of mechanism of drug resistance and development of overcoming strategy hold great significance in management of colon cancer. In this study, we discovered that activation of the PERK branch of the unfolded protein response (UPR) pathways is required for colon cancer cells to survive treatment of 5-Fluorouracil (5-FU), one of the first-line chemotherapeutics for late-stage colon cancer patients. Genetic and pharmacological inhibition of PERK or its downstream factors greatly sensitize colon cancer cells to 5-FU. Most importantly, in vivo use of PERK inhibitor synergizes with 5-FU in suppressing the growth of colon cancer cells in mouse models. In summary, our findings established a promising way to overcome resistance to chemotherapy in colon cancer. |
format | Online Article Text |
id | pubmed-6397152 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2019 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-63971522019-03-05 Activation of the PERK-ATF4 pathway promotes chemo-resistance in colon cancer cells Shi, Zhong Yu, Xiaofu Yuan, Meiqin Lv, Wangxia Feng, Tingting Bai, Rui Zhong, Haijun Sci Rep Article Colon cancer is a major health problem worldwide. While chemotherapy remains a main approach for treating late-stage colon cancer patients, most, if not all, of them will develop drug resistance and die of uncontrollable disease progression eventually. Therefore, identification of mechanism of drug resistance and development of overcoming strategy hold great significance in management of colon cancer. In this study, we discovered that activation of the PERK branch of the unfolded protein response (UPR) pathways is required for colon cancer cells to survive treatment of 5-Fluorouracil (5-FU), one of the first-line chemotherapeutics for late-stage colon cancer patients. Genetic and pharmacological inhibition of PERK or its downstream factors greatly sensitize colon cancer cells to 5-FU. Most importantly, in vivo use of PERK inhibitor synergizes with 5-FU in suppressing the growth of colon cancer cells in mouse models. In summary, our findings established a promising way to overcome resistance to chemotherapy in colon cancer. Nature Publishing Group UK 2019-03-01 /pmc/articles/PMC6397152/ /pubmed/30824833 http://dx.doi.org/10.1038/s41598-019-39547-x Text en © The Author(s) 2019 Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. |
spellingShingle | Article Shi, Zhong Yu, Xiaofu Yuan, Meiqin Lv, Wangxia Feng, Tingting Bai, Rui Zhong, Haijun Activation of the PERK-ATF4 pathway promotes chemo-resistance in colon cancer cells |
title | Activation of the PERK-ATF4 pathway promotes chemo-resistance in colon cancer cells |
title_full | Activation of the PERK-ATF4 pathway promotes chemo-resistance in colon cancer cells |
title_fullStr | Activation of the PERK-ATF4 pathway promotes chemo-resistance in colon cancer cells |
title_full_unstemmed | Activation of the PERK-ATF4 pathway promotes chemo-resistance in colon cancer cells |
title_short | Activation of the PERK-ATF4 pathway promotes chemo-resistance in colon cancer cells |
title_sort | activation of the perk-atf4 pathway promotes chemo-resistance in colon cancer cells |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6397152/ https://www.ncbi.nlm.nih.gov/pubmed/30824833 http://dx.doi.org/10.1038/s41598-019-39547-x |
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