Neuronal NO synthase mediates plenylephrine induced cardiomyocyte hypertrophy through facilitation of NFAT-dependent transcriptional activity

Neuronal nitric oxide synthase (NOS1) has been consistently shown to be the predominant isoform of NOS and/or NOS-derived NO that may be involved in the myocardial remodeling including cardiac hypertrophy. However, the direct functional contribution of NOS1 in this process remains to be elucidated....

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Detalles Bibliográficos
Autores principales: Loyer, Xavier, Dubroca, Caroline, Branchereau, Maxime, Griffith, Graziellia, Garcia, Luis, Heymes, Christophe
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2019
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6412025/
https://www.ncbi.nlm.nih.gov/pubmed/30899802
http://dx.doi.org/10.1016/j.bbrep.2019.100620

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6412025/
https://www.ncbi.nlm.nih.gov/pubmed/30899802
http://dx.doi.org/10.1016/j.bbrep.2019.100620

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