Deletion of p38α MAPK in microglia blunts trauma-induced inflammatory responses in mice

Traumatic brain injury (TBI) is a significant cause of morbidity and mortality in the USA and other developed countries worldwide. Following the initial mechanical insult, the brain’s primary innate immune effector, microglia, initiate inflammatory signaling cascades and pathophysiological responses...

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Detalles Bibliográficos
Autores principales: Morganti, Josh M., Goulding, Danielle S., Van Eldik, Linda J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2019
Materias:
Short Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6511220/
https://www.ncbi.nlm.nih.gov/pubmed/31077217
http://dx.doi.org/10.1186/s12974-019-1493-5

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6511220/
https://www.ncbi.nlm.nih.gov/pubmed/31077217
http://dx.doi.org/10.1186/s12974-019-1493-5

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