Protective Effects of a New C-Jun N-terminal Kinase Inhibitor in the Model of Global Cerebral Ischemia in Rats

c-Jun N-terminal kinase (JNK) is activated by various brain insults and is implicated in neuronal injury triggered by reperfusion-induced oxidative stress. Some JNK inhibitors demonstrated neuroprotective potential in various models, including cerebral ischemia/reperfusion injury. The objective of t...

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Detalles Bibliográficos
Autores principales: Plotnikov, Mark B., Chernysheva, Galina A., Aliev, Oleg I., Smol’iakova, Vera I., Fomina, Tatiana I., Osipenko, Anton N., Rydchenko, Victoria S., Anfinogenova, Yana J., Khlebnikov, Andrei I., Schepetkin, Igor A., Atochin, Dmitriy N.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2019
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6539151/
https://www.ncbi.nlm.nih.gov/pubmed/31058815
http://dx.doi.org/10.3390/molecules24091722

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6539151/
https://www.ncbi.nlm.nih.gov/pubmed/31058815
http://dx.doi.org/10.3390/molecules24091722

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