Augmented β2-adrenergic signaling dampens the neuroinflammatory response following ischemic stroke and increases stroke size

BACKGROUND: Ischemic stroke provokes a neuroinflammatory response and simultaneously promotes release of epinephrine and norepinephrine by the sympathetic nervous system. This increased sympathetic outflow can act on β2-adrenergic receptors expressed by immune cells such as brain-resident microglia...

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Detalles Bibliográficos
Autores principales: Lechtenberg, Kendra J., Meyer, Scott T., Doyle, Janelle B., Peterson, Todd C., Buckwalter, Marion S.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2019
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6540436/
https://www.ncbi.nlm.nih.gov/pubmed/31138227
http://dx.doi.org/10.1186/s12974-019-1506-4

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6540436/
https://www.ncbi.nlm.nih.gov/pubmed/31138227
http://dx.doi.org/10.1186/s12974-019-1506-4

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