Aβ-induced vulnerability propagates via the brain’s default mode network

The link between brain amyloid-β (Aβ), metabolism, and dementia symptoms remains a pressing question in Alzheimer’s disease. Here, using positron emission tomography ([(18)F]florbetapir tracer for Aβ and [(18)F]FDG tracer for glucose metabolism) with a novel analytical framework, we found that Aβ ag...

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Detalles Bibliográficos
Autores principales: Pascoal, Tharick A., Mathotaarachchi, Sulantha, Kang, Min Su, Mohaddes, Sara, Shin, Monica, Park, Ah Yeon, Parent, Maxime J., Benedet, Andrea L., Chamoun, Mira, Therriault, Joseph, Hwang, Heungsun, Cuello, A. Claudio, Misic, Bratislav, Soucy, Jean-Paul, Aston, John A. D., Gauthier, Serge, Rosa-Neto, Pedro
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2019
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6547716/
https://www.ncbi.nlm.nih.gov/pubmed/31164641
http://dx.doi.org/10.1038/s41467-019-10217-w

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6547716/
https://www.ncbi.nlm.nih.gov/pubmed/31164641
http://dx.doi.org/10.1038/s41467-019-10217-w

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