Preso regulates NMDA receptor-mediated excitotoxicity via modulating nitric oxide and calcium responses after traumatic brain injury

Traumatic brain injury (TBI) has become a major health concern worldwide, and the poor outcome of TBI increases the need for therapeutic improvement. Secondary injuries following TBI, including excitotoxicity, lead to synaptic dysfunction and provide potential targets for intervention. Postsynaptic...

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Detalles Bibliográficos
Autores principales: Luo, Peng, Li, Xin, Wu, Xiuquan, Dai, Shuhui, Yang, Yuefan, Xu, Haoxiang, Jing, Da, Rao, Wei, Xu, Hongyu, Gao, Xiangyu, Fei, Zhou, Lu, Hongbing
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2019
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6591282/
https://www.ncbi.nlm.nih.gov/pubmed/31235685
http://dx.doi.org/10.1038/s41419-019-1731-x

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6591282/
https://www.ncbi.nlm.nih.gov/pubmed/31235685
http://dx.doi.org/10.1038/s41419-019-1731-x

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