Cargando…
Tubular NOX4 expression decreases in chronic kidney disease but does not modify fibrosis evolution
BACKGROUND: NADPH oxidase 4 (NOX4) catalyzes the formation of hydrogen peroxide (H(2)O(2)). NOX4 is highly expressed in the kidney, but its role in renal injury is unclear and may depend on its specific tissue localization. METHODS: We performed immunostaining with a specific anti-NOX4 antibody and...
Autores principales: | Rajaram, Renuga Devi, Dissard, Romain, Faivre, Anna, Ino, Frédérique, Delitsikou, Vasiliki, Jaquet, Vincent, Cagarelli, Thomas, Lindenmeyer, Maja, Jansen-Duerr, Pidder, Cohen, Clemens, Moll, Solange, de Seigneux, Sophie |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6598841/ https://www.ncbi.nlm.nih.gov/pubmed/31247506 http://dx.doi.org/10.1016/j.redox.2019.101234 |
Ejemplares similares
-
Evolution of hypoxia and hypoxia-inducible factor asparaginyl hydroxylase regulation in chronic kidney disease
por: Faivre, Anna, et al.
Publicado: (2023) -
NADPH oxidase 4 deficiency increases tubular cell death during acute ischemic reperfusion injury
por: Nlandu-Khodo, Stellor, et al.
Publicado: (2016) -
Pharmacological characterization of the seven human NOX isoforms and their inhibitors
por: Augsburger, Fiona, et al.
Publicado: (2019) -
Tubular Cell Glucose Metabolism Shift During Acute and Chronic Injuries
por: Faivre, Anna, et al.
Publicado: (2021) -
Metabolic mechanisms of acute proximal tubular injury
por: Hall, Andrew M., et al.
Publicado: (2022)