TREM1/3 Deficiency Impairs Tissue Repair After Acute Kidney Injury and Mitochondrial Metabolic Flexibility in Tubular Epithelial Cells

Long-term sequelae of acute kidney injury (AKI) are associated with incomplete recovery of renal function and the development of chronic kidney disease (CKD), which can be mediated by aberrant innate immune activation, mitochondrial pathology, and accumulation of senescent tubular epithelial cells (...

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Detalles Bibliográficos
Autores principales: Tammaro, Alessandra, Scantlebery, Angelique M. L., Rampanelli, Elena, Borrelli, Cristiana, Claessen, Nike, Butter, Loes M., Soriani, Alessandra, Colonna, Marco, Leemans, Jaklien C., Dessing, Mark C., Florquin, Sandrine
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2019
Materias:
Immunology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6629955/
https://www.ncbi.nlm.nih.gov/pubmed/31354698
http://dx.doi.org/10.3389/fimmu.2019.01469

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6629955/
https://www.ncbi.nlm.nih.gov/pubmed/31354698
http://dx.doi.org/10.3389/fimmu.2019.01469

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