The Absence of Endothelial Sodium Channel α (αENaC) Reduces Renal Ischemia/Reperfusion Injury

The epithelial sodium channel (ENaC) has a key role in modulating endothelial cell stiffness and this in turn regulates nitric oxide (NO) synthesis. The physiological relevance of endothelial ENaC in pathological conditions where reduced NO bioavailability plays an essential role remains largely une...

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Detalles Bibliográficos
Autores principales: Tarjus, Antoine, González-Rivas, Cecilia, Amador-Martínez, Isabel, Bonnard, Benjamin, López-Marure, Rebeca, Jaisser, Frédéric, Barrera-Chimal, Jonatan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2019
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6651193/
https://www.ncbi.nlm.nih.gov/pubmed/31252520
http://dx.doi.org/10.3390/ijms20133132

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6651193/
https://www.ncbi.nlm.nih.gov/pubmed/31252520
http://dx.doi.org/10.3390/ijms20133132

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