Deficiency of Anoctamin 5/TMEM16E causes nuclear positioning defect and impairs Ca(2+) signaling of differentiated C2C12 myotubes

Anoctamin 5 (ANO5)/TMEM16E belongs to a member of the ANO/TMEM16 family member of anion channels. However, it is a matter of debate whether ANO5 functions as a genuine plasma membrane chloride channel. It has been recognized that mutations in the ANO5 gene cause many skeletal muscle diseases such as...

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Detalles Bibliográficos
Autores principales: Phuong, Tam Thi Thanh, An, Jieun, Park, Sun Hwa, Kim, Ami, Choi, Hyun Bin, Kang, Tong Mook
Formato: Online Artículo Texto
Lenguaje:English
Publicado: The Korean Physiological Society and The Korean Society of Pharmacology 2019
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6819897/
https://www.ncbi.nlm.nih.gov/pubmed/31680776
http://dx.doi.org/10.4196/kjpp.2019.23.6.539

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6819897/
https://www.ncbi.nlm.nih.gov/pubmed/31680776
http://dx.doi.org/10.4196/kjpp.2019.23.6.539

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