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Deficiency of Anoctamin 5/TMEM16E causes nuclear positioning defect and impairs Ca(2+) signaling of differentiated C2C12 myotubes
Anoctamin 5 (ANO5)/TMEM16E belongs to a member of the ANO/TMEM16 family member of anion channels. However, it is a matter of debate whether ANO5 functions as a genuine plasma membrane chloride channel. It has been recognized that mutations in the ANO5 gene cause many skeletal muscle diseases such as...
| Autores principales: | , , , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
The Korean Physiological Society and The Korean Society of Pharmacology
2019
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6819897/ https://www.ncbi.nlm.nih.gov/pubmed/31680776 http://dx.doi.org/10.4196/kjpp.2019.23.6.539 |