Sirtuin 3 deficiency does not impede digit regeneration in mice

The mitochondrial deacetylase sirtuin 3 (SIRT3) is thought to be one of the main contributors to metabolic flexibility–promoting mitochondrial energy production and maintaining homeostasis. In bone, metabolic profiles are tightly regulated and the loss of SIRT3 has deleterious effects on bone volume...

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Detalles Bibliográficos
Autores principales: Busse, Emily, Simkin, Jennifer, Marrero, Luis, Stewart, Kennon, Brunauer, Regina, Muneoka, Ken, Guntur, Anyonya, Lacey, Michelle, Sammarco, Mimi
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2019
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6848098/
https://www.ncbi.nlm.nih.gov/pubmed/31712596
http://dx.doi.org/10.1038/s41598-019-52921-z

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6848098/
https://www.ncbi.nlm.nih.gov/pubmed/31712596
http://dx.doi.org/10.1038/s41598-019-52921-z

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