EPO and TMBIM3/GRINA Promote the Activation of the Adaptive Arm and Counteract the Terminal Arm of the Unfolded Protein Response after Murine Transient Cerebral Ischemia

Ischemic stroke is known to cause the accumulation of misfolded proteins and loss of calcium homeostasis leading to impairment of endoplasmic reticulum (ER) function. The unfolded protein response (UPR) is an ER-located and cytoprotective pathway that aims to resolve ER stress. Transmembrane BAX inh...

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Detalles Bibliográficos
Autores principales: Habib, Pardes, Stamm, Ann-Sophie, Schulz, Joerg B., Reich, Arno, Slowik, Alexander, Capellmann, Sandro, Huber, Michael, Wilhelm, Thomas
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2019
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6862264/
https://www.ncbi.nlm.nih.gov/pubmed/31683519
http://dx.doi.org/10.3390/ijms20215421

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6862264/
https://www.ncbi.nlm.nih.gov/pubmed/31683519
http://dx.doi.org/10.3390/ijms20215421

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