Can N-Methyl-D-Aspartate Receptor Hypofunction in Schizophrenia Be Localized to an Individual Cell Type?

Hypofunction of N-methyl-D-aspartate glutamate receptors (NMDARs), whether caused by endogenous factors like auto-antibodies or mutations, or by pharmacological or genetic manipulations, produces a wide variety of deficits which overlap with—but do not precisely match—the symptom spectrum of schizop...

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Detalles Bibliográficos
Autores principales: Bygrave, Alexei M., Kilonzo, Kasyoka, Kullmann, Dimitri M., Bannerman, David M., Kätzel, Dennis
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2019
Materias:
Psychiatry
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6881463/
https://www.ncbi.nlm.nih.gov/pubmed/31824347
http://dx.doi.org/10.3389/fpsyt.2019.00835

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6881463/
https://www.ncbi.nlm.nih.gov/pubmed/31824347
http://dx.doi.org/10.3389/fpsyt.2019.00835

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