Ischemic Postconditioning Reduces Reperfusion Arrhythmias by Adenosine Receptors and Protein Kinase C Activation but Is Independent of K(ATP) Channels or Connexin 43

Ischemic postconditioning (IPoC) reduces reperfusion arrhythmias but the antiarrhythmic mechanisms remain unknown. The aim of this study was to analyze IPoC electrophysiological effects and the role played by adenosine A(1), A(2A) and A(3) receptors, protein kinase C, ATP-dependent potassium (K(ATP)...

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Detalles Bibliográficos
Autores principales: Diez, Emiliano Raúl, Sánchez, Jose Antonio, Prado, Natalia Jorgelina, Ponce Zumino, Amira Zulma, García-Dorado, David, Miatello, Roberto Miguel, Rodríguez-Sinovas, Antonio
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2019
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6928819/
https://www.ncbi.nlm.nih.gov/pubmed/31775376
http://dx.doi.org/10.3390/ijms20235927

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6928819/
https://www.ncbi.nlm.nih.gov/pubmed/31775376
http://dx.doi.org/10.3390/ijms20235927

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