Hypermethylation of mitochondrial DNA in vascular smooth muscle cells impairs cell contractility

Vascular smooth muscle cell (SMC) from arterial stenotic-occlusive diseases is featured with deficiency in mitochondrial respiration and loss of cell contractility. However, the regulatory mechanism of mitochondrial genes and mitochondrial energy metabolism in SMC remains elusive. Here, we described...

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Detalles Bibliográficos
Autores principales: Liu, Yue-Feng, Zhu, Juan-Juan, Yu Tian, Xiao, Liu, Han, Zhang, Tao, Zhang, Yun-Peng, Xie, Si-An, Zheng, Ming, Kong, Wei, Yao, Wei-Juan, Pang, Wei, Zhao, Chuan-Rong, Tang, Yuan-Jun, Zhou, Jing
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6971246/
https://www.ncbi.nlm.nih.gov/pubmed/31959742
http://dx.doi.org/10.1038/s41419-020-2240-7

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6971246/
https://www.ncbi.nlm.nih.gov/pubmed/31959742
http://dx.doi.org/10.1038/s41419-020-2240-7

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