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Inhibiting SLC26A4 reverses cardiac hypertrophy in H9C2 cells and in rats
BACKGROUND: It has been confirmed that mutations in solute carrier family 26 member 4 (SLC26A4) contribute to pendred syndrome. However, the role of SLC26A4 in cardiac hypertrophy and the signaling pathways remain unclear. METHODS: Cardiomyocytes were treated by 200 µM phenylephrine (PE) to induce c...
| Autores principales: | , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
PeerJ Inc.
2020
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6979409/ https://www.ncbi.nlm.nih.gov/pubmed/31998553 http://dx.doi.org/10.7717/peerj.8253 |