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TRPC6 inactivation does not protect against diabetic kidney disease in streptozotocin (STZ)‐treated Sprague‐Dawley rats

Canonical transient receptor potential‐6 (TRPC6) channels have been implicated in the progression of several forms of kidney disease (1). While there is strong evidence that glomerular TRPC6 channels are dysregulated in diabetic nephropathy (DN), there is no consensus as to whether deletion or inact...

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Detalles Bibliográficos
Autores principales: Hassanzadeh Khayyat, Naghmeh, Kim, Eun Young, Dryer, Stuart E.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2019
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6996301/
https://www.ncbi.nlm.nih.gov/pubmed/32123821
http://dx.doi.org/10.1096/fba.2019-00077